Are Causes Of Ben Liver Disease

The causative fungus is Pityrosporum orbiculare (Malassezia furfur), which parasitizes only in the epidermis of the epidermis, is benign but often recurs and affects the body's aesthetics.
EPIDEMIOLOGICAL CHARACTERISTICS OF LANG BEN
The causative agent of vitiligo
Pityrosporum orbiculare is a yeast-like, lipophilic and lipophilic fungus (keratin), living saprophytic on human skin. According to Robert, 97% of people have more pityrosporum ovale in the scalp than before puberty.
Healthy children <1 year of age usually do not find Pityrosporum but can be isolated on the skin of 37% of children hospitalized for some reason. The distribution of vitiligo The disease is widely distributed throughout the world, especially in the tropics and subtropics

. In temperate regions, the disease usually occurs in the summer (from May to September). The disease is found at all ages, mainly in young people (20 years), men are more ill than women. Favorable factors for vitiligo Conditions for the fungus to thrive on humans and turn to pathogens have not been clearly defined, but there are a number of favorable factors that are noted: Cases of increased mucus on the skin such as poor body hygiene, excessive sweating (occupation or climate), puberty, pregnancy, increased cortisone in the blood (systemic corticosteroid treatment or in Cushing's syndrome) , using cosmetics Impaired resistance due to malnutrition or immunosuppressive treatment in organ transplants ..


. can also cause an outbreak.
Genetic factors also seem to be related to the incidence of disease.
Mode of transmission of vitiligo
Directly from person to person, or indirectly through towels, clothes, bedding ..
Recently, the role of Pityrosporum orbiculare's systemic infection has also been mentioned in people, especially infants, who have been given intravenous lipids for a long time
SYMPTOMS OF LANG BEN
Vitiligo lesions
Lesions are very obvious inlaid, slightly woody, slightly flaky, when scraped, they will flake off fine flakes or leave white chalk-like marks (signs of shavings).
Lesions usually start from pores and spread into separate spots and then kill, many inlaid into large patches with crooked edges like a map. Basic lesions are hyperpigmented macules, milky milky or reddish-brown colors, which are clearly observed on white people or where the body is less exposed to sunlight such as the abdomen, breast, inner face. in the arm or thigh.



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However, in darker areas of the skin such as the back, on the outside of the arms, or in people with colored skin, the lesions appear as pigmented macules. The more you go out into the sun, the more the contrast between the pigments. The more you go out into the sun, the more pronounced the difference between a white, sick skin and tanned skin. In both patients, both types of lesions may appear
Lesions usually occur in the chest, upper back, c, upper back, shoulders, arms, abdomen, thighs, neck, front of the arms, very rarely in the scalp, palms, feet , never seen in body hair or nails. In some patients, the fungus may predominate in the inguinal folds.
Under the light of a Wood lamp, the fluorescence contrast is yellowish green.
The patient does not itch or itches little, but when sweating the patient may tingle tingling.
Acquired systemic infection (Catheter - acquired infection)
Met in newborns or adults who are receiving long-term treatment with intravenous lipids. In adults, the infection almost does not cause clinical manifestations, only by accident detected when blood transplants, or only prolonged fever.



. In contrast, manifestations of thrombocytopenia, leucoytosis with increased bands, are more common in infants.
IMMUNE WITH BEN LANG BEN
Over the age of 30, the incidence of the disease decreases, so it is assumed that there is acquired immunity, but the mechanism of immunogenesis is unknown
Almost all adults have an amount of IgG antibody to the Pityrosporum orbiculare in the blood.
PATIENT PHYSIOLOGY BEN
The primary lesion is hyperpigmentation which is caused by stimulated melanosomes, which increase the size and distribution of sporadic cells.
Subsequently, the lipid metabolism of the fungal cells produced several substances:
Toxicity to melanocytes reduces the synthesis of melanin.
Preventing the absorption of ultraviolet rays from sunlight through the skin should inhibit the production of melanin.
Dicarboxylic acid plays an inhibitory role in competition with tyrosinase
Therefore, normal skin when exposed to the sun is still affected by ultraviolet rays, so the pigmentation production increases while the lesions remain unchanged, becoming a clear contrast to the healthy skin.
Ultraviolet light of Wood lamp has a long wavelength λ, so it has low penetration capacity, it will fluoresce the ions of substances produced by pathogenic fungi.
DIAGNOSTIC DIAGNOSTIC BEN
Differential diagnosis of vitiligo
Tinea versicolor sometimes requires a differential diagnosis with the following cases:
Vitiligo (Vitiligo)
Pigmented eyes are not flaky but surrounded by hyperpigmentation.
The hair on the injured area is silvery white.




Sensitive to ultraviolet rays and prone to burning when exposed to sunlight.
Usually occurs in the face, left chest door, back of hands, armpits, cave, eyelids, nose.

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